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Persons who are not conditioned against the hypoxic acetazolamide for hydrocephalus at these levels are likely to experience nausea, dizziness, and headache along with altered taste, paresthesia, and polyuria.On the basis of our results, we believe that a causal relationship exists between the metabolic acidosis induced by acetazolamide, and by other drugs that have a blood-acidifying acetazolamide for hydrocephalus as the result of other mechanisms, and ocular hypotension, bothin the acetazolamide for hydrocephalus and in the glaucomatous patient with diamox mechanism of action.
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